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The 24th European Lipoprotein Club (ELC) Meeting will take place at the Evangelische Akademie, Tutzing, near Munich From September 10-13, 2001 Final Program Monday, September 10 20.00
State of the Art Lecture, Chair: H. Dieplinger
F. Lottspeich (Munich)
Proteomics – State of the Art Tuesday, September 11 Session I: Atherogenic Mechanisms, Chairs: M. Hofker,
D. Bowyer 8.30-9.15
S. Fazio (Nashville, TN) The arterial macrophage: So much more than a garbage
collector 9.15-9.35
L. Havekes (Leiden) The role of the tumour suppressor gene p53 in the
progression and
regression of atherosclerotic lesions 9.35-9.55
M. Gijbels (Maastricht) Genetic deletion of tissue plasminogen activator
(t-PA) in APOE3-Leiden mice reduces progression of cholesterol-induced
atherosclerosis 9.55-10.15
M. De Winther (Maastricht) Mouse model to study the role of snpPLA2 in
atherogenesis 10.15-10.45
Coffee break 10.45-11.05
N. Leitinger (Vienna) Influence of oxidised phospholipds on inflammatory
gene induction in endothelial cells 11.05-11.25 A. Stannard (London) Cell-derived apolipoprotein E stimulates nitric oxide
synthase (NOS) and inhibits vascular cell adhesion molecule-1 (VCAM-1) in
human endothelial cells 11.25-11.45
D. Milosavljevic (Paris) Elevated expression of LDL receptor related protein,
CLA-1 and SRA scavenger receptors in human monocyte-derived macrophages: role
in foam-cell formation 11.45-12.05
M. Klouche (Stuttgart) Expression of pentraxin-3 (PTX3) by human vascular
smooth muscle cells – synergistic action of degraded lipoproteins and
classic acute-phase proteins 12.30
Lunch 16.00-18.00
Wine & Science (Poster
Session) Session II: Regulation of cellular cholesterol
homeostasis, Chair: H. Dieplinger, P. Tarugi 19.30-20.00
R. Lawn (Palo Alto, CA) ABCA1: The gatekeeper for eliminating excess tissue
cholesterol 20.00-20.20
A. von Eckardstein (Münster) The ATP binding cassette transporter A1 contributes to
the secretion of interleukin-1b from macrophages, but not from monocytes 20.20-20.40
Y. Uehara (Münster) Regulation of the ATP binding cassette transporter A1
(ABCA1) by insulin 20.40-21.00
F. Kuipers (Groningen) Hepatic synthesis and hepatobiliary transport of
cholesterol are not affected in ABCA1-deficient mice 21.00-21.20 S. Lorkowski (Münster) The ATP-binding cassette transporter gene ABCG1 (ABC8)
is overexpressed in Tangier disease macrophages 21.20-21.40 C. Langer (Münster) Testerone stimulates selective cholesteryl ester
uptake in hepatocytes and efflux from macrophages 21.40-22.00
D. Delsing (Leiden) Expression of apolipoprotein C1 in macrophages reduces
cholesterol esterification and enhances cholesterol efflux Wednesday, September 12 Session III: Structure/function relationship of
lipoproteins Chair: P. Talmud, M. Jauhiainen 8.30-9.15
R.O. Ryan (Oakland, CA) Structural studies on helix bundle molecular switch
apolipoproteins 9.15-9.35
M. Baumstark (Freiburg) Structure of human LDL subfractions using X-ray
crystallography 9.35-9.55
G. Olivecrona (Umea) Studies of apolipoprotein C-II by NMR and
site-directed mutagenesis. Identification of residues in a helical region
important for activation of lipoprotein lipase 9.55-10.15
C. Labeur (Gent) Characterization of recombinant wildtype and
site-directed mutations of apolipoprotein C-III: lipid binding, displacement
of apoE and inhibition of lipoprotein lipase Coffee Break 10.45-11.00
M. Jauhiainen (Helsinki) The inactive form of human plasma PLTP: isolation and
partial characterization
11.00-11.15
F. Peelman (Gent) A hydrophobic cluster at the surface of the human
plasma phospholipid transfer protein is critical for activity on high density
lipoprotein substrates 11.15-11.35
G. Sperber (London) Chimeraplasts as reagents to probe structure-function
relationships in ApoA-I and LCAT 11.35-11.55
P. Kovanen (Helsinki) Chymase proteolyzes apoA-I in discoidal reconstituted
HDL particles by cleaving the amino- and carboxy-termini: implications for the
mechanism of cellular cholesterol efflux induced by lipidated apoA-I 12.30 Lunch Session IV: Gene regulation in atherosclerosis, Chair:
H. Funke, B. Staels 14.00-14.45
B. Staels (Lille) Nuclear receptors controlling lipid metabolism and
inflammation in the vascular wall 14.45-15.05
B. Weitkamp (Münster) Gene expression with defined cell populations
collected from atherosclerotic plaques by laser capture microdissection and
quantification of gene expression by real-time PCR 15.05-15.25
A. Kreeft (Leiden) Expression profiling to identify novel dietary
response genes involved in lipid metabolism 15.25-15.55
Coffee Break 15.55-16.15
A. Verhoeven (Rotterdam) The hepatic lipase 514 C->T polymorphism interferes
with the stimulation of gene expression by NEFA‘s 16.15-16.35
S. Humphries (London) Functional effects of interleukin-6 (IL6) promoter
polymorphisms in vivo and in vitro 16.35-16.55
G. Tan (Oxford) Subcutaneous adipose tissue function in a subject with
a dominant negative PPARg mutation (PPARgP467L) 16.55-17.15
J. Chinetti (Lille) PPAR-alpha controls cholesterol homeostasis in human
monocyte-derived macrophages 20.00
Social Event Thursday, September 13 Session V: Insulin resistance, NEFA’s and obesity,
Chair: J. Nimpf, G. Dallinga-Thie 9.00-9.45
R. Farese (San Francisco, CA) Triglyceride synthesis and obesity: insights from DGAT-deficient
mice 9.45-10.05
D. Chirieac (Rochester, NY) Selective resistance to insulin-mediated supression of
very low density lipoprotein production in Zucker diabetic fatty rats 10.05-10.25
J. Strauss (Graz) EL mediates HDL binding, particle and selective uptake
in HepG2 cells 10.25-10.45 G.
Hämmerle (Graz) The in vivo role of hormone-sensitive lipase (HSL) in
lipid mobilisation 10.45-11.05 P.
Voshol (Leiden) No altered glucose uptake but increased glucose
oxidation in hormone sensitive lipase knockout mice 11.05-11.35
Coffee break and Young Investigator award 11.35-11.55
F. Van’t Hooft (Stockholm) Human evidence that the apolipoprotein A-II gene is
implicated in visceral fat accumulation, metabolism of triglyceride-rich
lipoproteins and protection against early atherosclerosis 11.55-12.15 J.
Björkegren (San Francisco, CA) Microsomal triglyceride transfer protein and hepatic
toxicity 12.30 Lunch
+++++++++++++++++++++++++++++++++++++++++++++++++++++++++++ If you wish to attend this year's Meeting, please complete the abstract form as shown below and return to: Hans Dieplinger 1) FIND AN EXAMPLE OF THE ABSTRACT FORM AND THE GUIDELINESS FOR SUBMISSION BELOW 3) MAIL THE COMPLETED FORM TO OUR SECRETARY (also FAXES, OR E-MAIL FORMS ACCEPTABLE, but regular mail is still preferred) ________________________example - do not copy this form _________________________________________ 24th ELC Meeting September 10-13, 2001 Deadline May 1st, 2001!!! Applicant (use separate form for each applicant): Family Name:.......................................... First Name:..............................Sex: F / M Address:......................................................................................................................... Telephone number:...............................Faxnumber:............................................... e-mail (obligatory):.................................................................................................... This abstract is for session number I / II / III / IV/other Other (please circle) Abstract of proposed presentation (keep the length of the abstract within this page (i.e. one page A4 format) for all text including the heading above)
Please include here 4-5 key words describing your research interest. ......................................................................................................................... ____end of page_______________________________________________
EUROPEAN LIPOPROTEIN CLUB GUIDELINES FOR SUBMISSION OF ABSTRACTS The primary purpose of the ELC is to promote discussion and exchange of ideas on the chosen topics. In recent years the number of applications for the ELC has increased considerably, but because of the size of the lecture room and the accommodation available, the number of people attending the conference is strictly limited. Because applicants are chosen on the basis of the abstracts they submit, the following guidelines have been drawn up by the Committee. 2. All abstracts are rated/graded by the Committee members, and applications are exclusively chosen on the scientific merit of the work. Abstracts not strictly related to the topics will be considered for possible participation in a varia session. 3. Only a few of the chosen applicants will be invited to present their work, but all those invited are expected to participate in the general discussion of the presentations. 4. The Committee feels that discussion and exchange of ideas is best promoted by having people with a wide range of experience and expertise at the Meeting, and that as many different laboratories should by represented. Because of this, in general, some of multiple applications from a single laboratory are unlikely to be rated highly. 5. The Committee also feels that good discussion is promoted by having the widest possible range of countries represented at the conference. For this reason, where similar abstracts are competing for a place, consideration to country of origin may be taken into account. 6. Because of the timing of
the grading and selection procedure, abstracts received after the deadline
of 1st May will not be accepted. 7. The selection of abstracts takes place at a Committee meeting in June, and those selected will be informed soon after by the Secretary. Those selected to give an oral presentation of their work will be contacted by the session Chairpersons. 8. Selected applicants are expected to stay for the full length of the meeting. 9. A "waiting list" of applicants, whose abstracts scored below the cut-off point, is kept by the Secretary. If any places become vacant, individuals on this list will be approached. 10. All abstracts are treated as confidential by the Committee members and are not published. However, after the meeting in September the session Chairpersons prepare a report, based on the presentations, which will be published in Atherosclerosis. If this creates a conflict for any of the speakers, they should discuss the situation with the Chairpersons of their session beforhand
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